Age groups you are accepting new hip dysplasia patients: Infants and children, Adolescents, Adults
Name: Jonathan Schoeneckier
Degree: MD, PhD
Title / Position: Assistant Professor of Orthopaedics, Pharmacology, Pediatrics and Pathoology
Place of Employment / Office Name: Vanderbilt University Medical Center
Street Address: 4202 Doctor’s Tower, 2200 Children’s Way
Zip/Postal code: 37232-9565
Country: United States
Office phone number: 615.343.5875
Office fax number: 615.343.2423
Medical or Surgical Specialty: Pediatric Orthopaedics
Professional Education and Memberships
Medical School: Duke University
Residency: VAnderbilt University
Fellowship training:Boston University (Boston Children’s Hospital)
Certifications:American Board of Orthopaedic Surgeons
Society Memberships (list up to four only):
Pediatric Orthopaedic Society of North America
American Academy of Orthopaedic Surgeons
Orthopaedic Research Society
Specific training in hip dysplasia:Fellowship training at Boston Children’s HospitalNumerous courses
Academic contributions for DDH research or education including publications:
OKU 11- authored pediatric hip (including DDH)
Cole HA, Yuasa M, Hawley G, Cates JM, Nyman JS, Schoenecker JG. Differential Development of the Distal and Proximal Femoral Epiphysis and Physis in Mice. Bone. 2012 Oct 15, PMID:2307913916.
Sankar WN, Schoenecker JG, Mayfield ME, Kim YJ, Millis MB Acetabular retroversion in down syndrome. J Pediatr Orthop. 2012 Apr;32(3):277-81.17.
Schoenecker JG Pathologic Hip Morphology in Cerebral Palsy and Down Syndrome J Pediatr Orthop. Accepted18.
Desai AA, Martus JE, Schoenecker J, Kan JH Spica MRI after closed reduction for developmental dysplasia of the hip. Pediatr Radiol. 2011 Apr;41(4):525-9. Epub 2011 Jan 29
Optional Personal Statement: I am a pediatric orthopaedic surgeon with a strong interest in defining the role of the vascular system in muscle and bone biology. My unique focus stems from my surgical training in musculoskeletal diseases in combination with my basic science training in vascular and bone biology. I have directed my basic and clinical research from observations in my clinical practice and laboratory combined with historical work defining the role of vascularity in muscle and bone biology. The underlying theme of our research is that all musculoskeletal tissue develops and regenerates in response to the availability of oxygen delivered by an intricate and essential vascular network. We postulate that there are critical molecular mechanisms within muscle and bone designed to sense and resolve hypoxia. As such, the overarching goals of our research is to define these mechanisms and develop novel treatments designed to improve pathological conditions resulting from conditions that disrupt the balance of musculoskeletal development, regeneration and cancer. As such, I have focused my clinical research on pathologic processes of muscle and bone with a presumed vascular etiology such as; DDH (development and repair of AVN) Legg-Calve-Perthes disease, Duchene’s muscular dystrophy, avascular necrosis, fracture non-union, slipped capital femoral epiphysis, osteosarcoma and infection induced bone necrosis. The goal of these studies is to define how changes in vascularity imposes subsequent pathology in in muscle and bone biology with the long term hope of addressing the vascular insult to prevent or treat these devastating diseases in adults and children.